Hai fellow physiotherapists....this blog basically contains Amal's compilation of exam preparation materials for NPTE, PCE, HAAD, DHA,MOH, PROMETRIC, ACOPRA.

Tuesday, June 23, 2015

Neurological dysfunctions

Question
Answer
Inflammation of membranes of SC or brain
Meningitis
Which meningitis is usually sicker with more rapid time course
Bacterial
Infection and inflammation of brain
Encephalitis
S/S of brain abscess
HA, fever, brainstem compression, Focal signs in CNII & CNVI
AIDS dementia syndrome (ADC)
confusion, memory loss, disorientation, ataxia, weak, tremor, hypersensitivity, pain, sensory loss
Formation of blood clot or thrombus within cerebral arteries or branches
Cerebral thrombosis
Traveling bits of matter that produce occlusion & infarction in cerebral arteries
Cerebral embolism
Abnormal bleeding as a result of rupture of blood vessel
Cerebral hemorrhage
Risk factors for CVA
atherosclerosis, HTN, CD, DM2, TIA
Lack of oxygen to brain
Cerebral anoxia
Irreversible cellular damage to brain
Cerebral infarction
Accumulation of fluids within brain
Cerebral edema
ICA syndromes include
ACA syndrome and MCA syndrome
ACA supplies
Supplies anterior 2/3 of medial cerebral cortex.
ACA syndrome
CL sensory loss, CL hemiparesis, leg > arm.
Occlusions proximal to anterior communicating artery produce
minimal deficits due to Circle of Willis
MCA supplies
lateral cerebral cortex, basal ganglia, internal capsule
MCA syndrome
CL sensory loss, CL hemiparesis arm>leg, Broca’s aphasia, perceptual dysfunction, homonymous hemianopsia, CL los of conjugate gaze, sensory ataxia
Medial medullary syndrome
VBA occlusion – IL tongue paralysis, CL paralysis of arm and leg, decr sensation
Lateral medullary syndrome (Wallenberg’s)
IL cerebellar, Horner’s syndrome, dysphagia, impaired speech, decr gag reflex, IL arm/trunk/leg sensory loss, CL pain/temp loss
Horner’s syndrome
miosis, ptosis, decr sweating
Basilar artery syndrome
brain stem S/S and PCA s/s. Locked in syndrome
Locked in syndrome
basilar artery occlusion at level of pons. Preserved consciousness but quadriplegia, anarthria.
Medial inferior Pontine syndrome
IL symptoms: cerebellar, conjugate gaze paralysis, diplopia. CL symptoms: hemiparesis, decr sensation.
Lateral inferior pontine syndrome
IL: cerebellar, facial paralysis, conjugate gaze paralysis, deafness, tinnitus. CL: pain/temp sensation
PCA syndrome
CL homonymous hemianopsia, CL sensory loss, thalamic syndrome, involuntary mvmts, CL transient hemiparesis, Weber’s syndrome, visual s/s.
Weber’s syndrome
oculomotor nerve palsy with CL hemiplegia
Lesions of parieto-occipital cortex of dominant hemisphere lead to
aphasia
Lesions of parietal lobe of non-dominant hemisphere lead to
perceptual deficits
Pt with lesion of L hemisphere (R hemi) tend to be
slow, cautious, hesitant, insecure
Pt with lesion of R hemisphere (L hemi) tend to be
impulsive, quick, indifferent, poor judgement, overestimate abilities, underestimate problem
L hemisphere lesion pts learning guidelines
appropriate communication method, frequent feedback & support, do not underestimate learning ability
R hemisphere lesion pts learning guidelines
use verbal cues, demo may confuse, give frequent feedback, focus on slow & control, avoid spatial clutter, do not over estimate ability to learn

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