Hai fellow physiotherapists....this blog basically contains Amal's compilation of exam preparation materials for NPTE, PCE, HAAD, DHA,MOH, PROMETRIC, ACOPRA.

Monday, May 25, 2015

Basics of ECG- How different factors show up in ECG.

Question
Answer
P wave
atrial DEpolarization
PR interval
time required for impulse to travel from atria through conduction system to Purkinje fibers
QRS wave
ventricular DEpolarization
ST segment
beginning of ventricular REpolarization
T wave
ventricular REpolarization
QT interval
time for electrical systole
Heart Rate
# of intervals between QRS complexes in a 6-second strip multiplied by 10
Ventricular arrhythmias
originate from an ectopic focus in the ventricles (outside normal conduction system)
Ventricular fibrillation
pulseless, emergency situation requiring EMT: CPR, defibrillation, medications
Premature ventricular contractions (PVCs)
NO P wave, bizarre & wide QRS that is premature, followed by a long compensatory pulse
Serious PVCs
>6 per minute, paired or in sequential runs, multifocal, very early PVC (R on T Phenomena)
Ventricular tachycardia
3 or more PVCs occuring sequentially; very rapid rate (150-200 bpm)
Ventricular tachycardia
wide, bizarre QRS waves, NO P waves, seriously compromised cardiac output
Ventricular fibrillation
chaotic activity of ventricle originating from multiple foci; unable to determine rate
Ventricular fibrillation
bizarre, erratic activity without QRS complex
Ventricular fibrillation
No effective cardiac output; clinical death within 4-6 mins
Atrial arrhythmias (supraventricular)
Rapid & repetitive firing of 1 or more ectopic foci in the atria
Atrial arrhythmias (supraventricular)
P waves abnormal (variable in shape) or not identifiable (atrial fibrillation)
Atrial tachycardia
140-250 bpm
Atrial flutter
250-350 bpm
Atrial fibrillation
>300 bpm
Atrioventricular blocks
ab(N) delays or failure to conduct through (N) conductiong system
If ventricular rate is slowed,
Cardiac output is decreased
3rd degree AV block
life threatening, requires meds (ATROPHINE), pacemaker
ST depression
impaired coronary perfusion (ischemia or injury)
MI central zone of infarction
Ab(N) Q waves
MI zone of injury
ST elevation
MI zone of ischemia
T wave inversion
Hyperkalemia
wide QRS, flat P wave, peaked T wave
Hypokalemia
flat T wave (or inverted), produces U wave
Hypercalcemia
wide QRS, short QT interval
Hypocalcemia
prolonged QT interval
Hypothermia
ST segment elevation; slow rhythm
Digitalis
ST segment depression, flattened T wave (or inverted), shortened QT
Quinidine
long QT, T flat/inverted, QRS long
BetaBlockers (Propranolol/Inderal)
decreased heart rate, blunts HR response to exercise
Nitrates (nitoglycerin)
Inc HR
Antiarrhythmic agents
may prolong QRS & QT intervals

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